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p -values lower than 0.05 were considered to be statistically significant
Moreover, AMPK and mTORC1 play opposing roles in controlling cellular metabolism, and the activity of mTORC1 is suppressed by AMPK through a dual mechanism involving the phosphorylation-induced activation of tuberous sclerosis complex 2 (TSC2) 162 and the phosphorylation-mediated inhibition of regulatory-associated protein of mTOR (Raptor), a component of the mTORC1 complex
Cardiovascular actions of the ghrelin gene-derived peptides and growth hormone-releasing hormone
Hypoxia and reoxygenation induce endothelial nitric oxide synthase uncoupling in endothelial cells through tetrahydrobiopterin depletion and S-glutathionylation
