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TB-500, an actin-binding protein, assists in sequestering actin where it is most beneficial for cell movement [2]

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MiR-3926 Inhibits Synovial Fibroblasts Proliferation and Inflammatory Cytokines Secretion Through Targeting Toll Like Receptor 5

In two Phase 2 clinical trials covering stasis and pressure ulcers, TB4 accelerated complete wound closure by almost a month in patients who achieved full healing representing one of the most substantial healing acceleration outcomes observed for a peptide compound in a controlled clinical investigation