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That said, we still need more detailed human studies to fully clarify these questions
The model tests biological plausibility, not clinical causation.Why noteworthy: it offers a mechanistic framework for studying timing, sex, and interacting developmental risks without proving that THC alone causes schizophrenia
It sits between the 8mg and 12mg levels tested in phase 2, offering a middle ground for individuals who find 12mg difficult to tolerate but want more effect than 8mg provides
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