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The lack of GSH detection can be explained by the possibility that free GSH is maintained in apoplast at a minimum level and is transported back to cytosol after GSSG is reduced
Diagnosis usually entails measuring serum levels of vitamin B12 or identifying elevated levels of methylmalonic acid and homocysteine
BMC gastroenterology , 17 (1), 96
Recent studies suggest that genetic susceptibility plays a crucial role in the coexistence of these conditions, with overlapping genetic variants influencing both vascular homeostasis and tumorigenesis
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