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This paradoxical condition fulfils two fundamental tasks: (a) it provides the biosynthesis of FA important for cancer propagation, while ensuring an important source of ATP and Nicotinamide adenine dinucleotide (NADH) by the catabolism of any FA excess

doi: 10.1111/dom.2016.18.issue-3 28 LiZYZhangNWenSZhangJSunXLFanXMet al

Furthermore, many researchers have found that reduced GSH levels and GSH-dependent antioxidant enzyme activity in damaged myocardium due to excess ROS and disruption of the GSH synthesis process, along with increased oxidative damage and ferroptosis