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This approach maintains the natural IGFBP-binding architecture and pituitary feedback regulation, avoiding the risks associated with direct exogenous IGF-1 receptor stimulation [19]
Reynolds, MS, PhD, Kimberly M
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An imbalance in the ratio between reduced (GSH) and oxidised (GSSG) forms of glutathione is implicated in various enterovirus infections 23 and depletion of cellular GSH levels using the GSH biosynthesis inhibitor L-buthionine sulfoximine (BSO) 24,25 and the small molecule inhibitor TP219 26 blocks the assembly and morphogenesis of many enterovirus capsids
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