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Tahiliani and colleagues revealed the therapeutic value of MO leaf extract in the regulation of hyperthyroidism, an auto-immune related disorder, by inhibiting triiodothyronine (T3) synthesis and release (Tahiliani and Kar, 2000)
The mtDNA is more vulnerable to excessive ROS-related damage to biomolecules, such as DNA repair enzyme proteins, than nuclear DNA (112), which disrupts the ETC and ultimately induces dysregulation of mitochondrial OXPHOS (113)
But that misses the point entirely
Extended Data Figure 8 Structures of class B GPCR ligands bound to NTDs
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