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Thazhath SS, Wu T, Bound MJ, et al

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Skoulidis F, Arbour KC, Hellmann MD, Patil P, Marmarelis ME, Owen D, Swad MM, Murray J, Levy B, Hellyer J, Gainor JF, Stewart T, Goldberg S, Dimou A, Bestvina C, Cummings AL, Elamin YY, Lam VK, Zhang J, Shu CA, Riess JW, B lakely C Pecot C, Mezquita L, Tabbo F, Sacher A, Scheffler M, Bicciuti B, Venkatraman D, Rizvi H, Liu C Johnston RP, Ni Y, Azok J, Kier M, Katz S, Davies K, Segal J, Ritterhouse L, Shaish H, Laroix L, Memmott RM, Madrigal JR, Goldman JW, Lau S, Carter B, Woodcock M, Roth JA, Swisher SG , Leighl N, Wolf J, Scagliotti GV, Planchard D, Besse B, Bivona T, Gandara DR, Garon EB, Rizvi N, Camidge R, Schalper K, Herbst RS, Shaw A, Neal J, Wakelee HA, Brahmer J, Janne P, Carbone DP, Aggarwal C, Pennell N, Rudin C, Papadimitrakopoulou V, Heymach JV

Excessive APAP metabolism depletes glutathione and increases N-acetyl-p-benzoquinoneimide (NAPQI) levels, leading to oxidative stress, DNA damage, and cell necrosis in the liver, which in turn leads to liver damage