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This is sustained by several studies indicating the presence of AGEs in the periphery of Lewy bodies, SNpc, cerebral cortex and amygdala of PD patients brains, suggesting that protein glycation is part of PD pathogenic mechanisms (Kurz et al., 2011)
Methylcobalamin form is the active form of vitamin B12 and does not require conversion in the body to be used
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Genome-wide loss of 5-hmC is a novel epigenetic feature of Huntington's disease
