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In laboratory settings, ghk-cu peptide is studied for its role in copper-dependent peptide interactions, gene-expression signaling, and tissue-associated molecular pathways under controlled experimental conditions
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Considering that cuproptosis-related proteins DLAT, LIAS, FDX1, and lipoylated mitochondrial enzymes are in mitochondria, the treatment of MAD, mitoCPR and mitoTAD outside mitochondria and on the surface of mitochondria may not play a role
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