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The results showed that the transient expression of SsCm1 inhibited callose deposition and the expression of pathogenesis-related genes ( NbPR1a and NbPR2 ) induced by PAMPs, including Flg22, chitin, and Sclerotinia culture filtrate elicitor (SCFE), while no significant variation was detected in the ROS burst induced by PAMPs (Supplementary Fig

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This phenomenon occurs due to an imbalance between the bodys antioxidant defence mechanisms and the production of reactive oxygen species (ROS)