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Description

Investigation of an albumin-enriched fraction of human serum and its albuminome

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The methionine loading results in an abnormal increase in homocysteinemia, revealing a defect in the transsulfuration pathway, which is attenuated by folic acid and/or pyridoxine supplementation

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Another possibility is that the conformational change induced by ipamorelin at the GHS-R1a receptor produces a biased signaling profile that activates GH release pathways without engaging pathways linked to ACTH secretion

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