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It has been shown that in patients with type 2 diabetes mellitus (T2DM) the response to the incretin effect is altered as a result of a severe defect in the sensitivity for GIP in the cells [4] and a reduction in food-induced GLP-1 secretion [5, 6]
In the pancreas, these agents enhance glucose-dependent insulin secretion while suppressing inappropriate glucagon release, improving glycemic control with a low risk of hypoglycemia when used alone (though this risk increases when combined with insulin or sulfonylureas)
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