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In contrast, another metabolomic study of tracheal aspirates from infants 28 weeks gestation noted that early decreases in fatty-acid metabolism, particularly the fatty acid -oxidation pathway, may predispose infants to developing BPD 46

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5A, lane 4), suggesting that Ube2e2 that interact with DET1 may facilitate the initial modification of c-Jun with COP1 RING domain