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The broad substrate specificity of GSTs allows them to protect cells against a range of toxic chemicals, and a lack of these enzymes is a disadvantage for the susceptible individual (Salinas and Wong 1999) whose risk may increase in many biochemical pathways

In vitro studies have shown that addition of DPP-4 is associated with impaired insulin signaling in primary human adipocytes, skeletal muscle, and smooth muscle cells (79), while conversely, silencing Dpp4 (using short interfering RNA) resulted in improved adipocyte insulin responsiveness (89)

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