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GSH prevents LOsG-induced pancreatic fibrosis via the TGF/SMAD pathway in vivo Considering the crucial role of TGF-1 in organ fibrosis, we examined whether LOsG contributed to TGF-1 expression by detecting TGF-1 protein levels in pancreatic tissues using western blotting
Sex and gender differences in risk, pathophysiology and complications of type 2 diabetes mellitus
Consistent with transcriptomic suppression of glycolytic and mitochondrial pathways, melatonin-treated ILC2s exhibited marked functional alterations in glucose utilization and oxidative metabolism ( Figures 5GS )
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