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This drastically reduces sublingual absorption and forces most of the medication through the GI tract, where bioavailability plummets
2.2 Neuroinflammation in the brain and cognitive dysfunction in diabetes Neuroinflammation itself is a defence mechanism against acute the CNS damage and ameliorates the effects of toxic substances produced in brain nerve cells, but persistent neuroinflammation inhibits nerve regeneration, leading to neurodegeneration making the patient cognitively impaired
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