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Abstract Introduction: Sepsis-associated encephalopathy (SAE) is characterized by acute neurological dysfunction and hippocampal damage, with oxidative stress being a key driver of neuronal injury
It is worth noting that, nuclear GSTP inhibited the association between HMGB1 and cPKC and reduced cPKC-mediated HMGB1 phosphorylation through interacting with HMGB1 in response to LPS stimulation
WangMWangMZhuQYaoXHeizhatiMCaiXet alDevelopment and validation of a coronary heart disease risk prediction model in snorers with hypertension: a retrospective observed study
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