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For example, the rapid pyroptotic cell death of SARS-CoV-2-infected blood monocytes or lung-resident macrophages prevents the virus from completing its reproductive cycle ( However, DAMPs-mediated signalling can also generate detrimental effects, particularly when a massive release of pro-inflammatory DAMPs from dying cells causes more cells to die: a situation that strongly contributes to the CD4+ T cell depletion in patients infected with HIV-1 (63, 64), reviewed in (65)