[19] Because of its antioxidant role, GSS mostly produce GSH inside the cytoplasm of liver cells and imported to mitochondria where detoxification occurs
PARP1-dependent kinetics of recruitment of MRE11 and NBS1 proteins to multiple DNA damage sites
This lipid mediator also reduced IL-1, IL-6 and metalloproteinase 3 expression in synovial fibroblasts, while increasing the metalloproteinase inhibitor TIMP1 (147)
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