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It works by stimulating the production of growth factors and increasing blood flow to damaged tissues
When I raised this with my GP, his response was extremely negative (likening GLP-1s to the next thalidomide and dismissing online sources as fraudulent)
After stroke, NPCs have the ability to migrate to the lesion site, and chemokines and IGF-1 can promote NPCs proliferation, migration, and differentiation (101, 102)
No doubt the fact that surgical anesthetics were being widely used by this time had something to do with this
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