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Blocking GSH synthesis in combination with JMF4073 depletes dTTP/dCTP pools to eradicate T315I-Bcr-Abl leukemia We hypothesize that reducing the GSH level to increase replication stress can render T315I-Bcr-Abl leukemia more susceptible to JMF4073 in vivo
Thus, adherence to treatment is causally linked with improved glycemic control and vice versa
ascorbate, is not very effective, probably due to its possible prooxidant behavior under certain conditions (e.g
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