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Another rat study found that acute restraint stress can upregulate TLR4 gene expression in the frontal cortex by inducing gut microbiota translocation, while intervention with antibiotics or the TLR4 specific inhibitor TAK-242 effectively suppresses this process and reduces the accumulation of inflammatory and oxidative/nitrosative mediators (163)

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however, the association of these enzymes to the erythrocyte membrane has been reported in different in vivo and in vitro studies [26, 31, 32, 93, 94, 95, 96, 97]

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