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In cancer cells, a heightened GSH level is indispensable for scavenging excessive ROS and detoxifying xenobiotics, rendering it a potential target for cancer therapy [9]

However, a negative result for those antibodies is of limited value, since some patients with PA lack the antibodies

Furthermore, while JNK and its downstream targets c-Jun and ATF2 were activated during the early steps of the liver regeneration in wild-type animals, the constitutively active JNK found in the quiescent liver of Gstp1/2 knockout mice underwent a decrease in its activity after PH